Oxygen-Dependent Accumulation of Purine DNA Lesions in Cockayne Syndrome Cells

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Reduced RNA polymerase II transcription in extracts of cockayne syndrome and xeroderma pigmentosum/Cockayne syndrome cells.

The hereditary disease Cockayne syndrome (CS) is a complex clinical syndrome characterized by arrested post-natal growth as well as neurological and other defects. The CSA and CSB genes are implicated in this disease. The clinical features of CS can also accompany the excision repair-defective hereditary disorder xeroderma pigmentosum (XP) from genetic complementation groups B, D or G. The XPB ...

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Repair of 8-oxoguanine in DNA is deficient in Cockayne syndrome group B cells.

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DNA damage during the G0/G1 phase triggers RNA-templated, Cockayne syndrome B-dependent homologous recombination.

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ژورنال

عنوان ژورنال: Cells

سال: 2020

ISSN: 2073-4409

DOI: 10.3390/cells9071671